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For decades, scientists have been intrigued by tiny viruses whose genetic material can be read both forward and backward. New research begins to explain this puzzling property.
Cells in symbiotic partnership, sometimes nested one within the other and functioning like organelles, can borrow from their host’s genes to complete their own metabolic pathways.
Maintaining perfect stability through negative feedback is a basic element of electrical circuitry, but it’s been a mystery how cells could do it — until now.
Viruses and other parasites may sync with their host’s biological clock — or reset it — to gain an advantage.
For 50 years, evolutionary theory has emphasized the importance of neutral mutations rather than adaptive ones at the level of DNA. Real genomic data challenges that assumption.
Using a new CRISPR-based technique, researchers are examining how the position of DNA within the nucleus affects gene expression and cell function.